Sodium Molybdate
Essential cofactor for the four molybdoenzymes: xanthine oxidase, aldehyde oxidase, sulphite oxidase, and mitochondrial amidoxime reducing component. Required for normal sulphur amino acid catabolism and purine metabolism. 50mcg = 100% NRV.
Mechanism
Molybdenum is incorporated as the molybdenum cofactor (Moco) into four essential mammalian enzymes: xanthine oxidase/dehydrogenase (purine catabolism to uric acid); aldehyde oxidase (oxidation of aldehydes and heterocycles, including drug metabolism); sulphite oxidase (converting sulphite to sulphate - the final step in sulphur amino acid catabolism, essential for metabolising cysteine and methionine); and mitochondrial amidoxime reducing component (mARC).
The most clinically critical of these is sulphite oxidase - genetic molybdenum cofactor deficiency results in severe neurological damage from sulphite accumulation. While dietary deficiency is rare in adults, ensuring adequate intake completes the full mineral cofactor profile required for normal xenobiotic and sulphur metabolism.
Key Benefits
- Cofactor for sulphite oxidase - essential for sulphur amino acid catabolism
- Required for xanthine oxidase (purine metabolism)
- Required for aldehyde oxidase (drug and xenobiotic metabolism)
- Completes the full molybdoenzyme set
- 50mcg = 100% NRV
Sodium Molybdate
75mcgSodium molybdate - pharmaceutical grade
Any time of day. Molybdenum is well-absorbed regardless of timing or food matrix. 50mcg = 100% NRV.